STEPHAN Delphine
Onglets principaux
Publications
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Mutations in specific domains of MT5-MMP prevent the accumulation of toxic APP metabolites and serve as templates for peptide-based therapeutics in human in vitro models of Alzheimer's disease2024
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Suppression of MT5-MMP reveals early modulation of Alzheimer’s pathogenic events in primary neuronal cultures of 5xFAD mice2024
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MT5-MMP promotes neuroinflammation, neuronal excitability and Aβ production in primary neuron/astrocyte cultures from the 5xFAD mouse model of Alzheimer’s diseaseJournal of Neuroinflammation2022
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The Alzheimer’s disease risk factor APOE4 drives pro-inflammation in human astrocytes via HDAC-dependent repression of TAGLN32022
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The C-terminal of MT5-MMP regulates C99 processing and Aβ levels in a cell model of Alzheimer's disease2022
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APOE4 primes inflammation in human astrocytes through the downregulation of a newly discovered target candidatevv2021
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TWEAK and TNFα, Both TNF Ligand Family Members and Multiple Sclerosis-Related Cytokines, Induce Distinct Gene Response in Human Brain Microvascular Endothelial CellsGenes2022
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APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activationCell Reports2022
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APOE4 drives inflammation in human astrocytes via TAGLN3 repression and NF-κB activationCell Reports2022
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MT5-MMP promotes neuroinflammation, neuronal excitability and Aβ production in primary neuron/astrocyte cultures from the 5xFAD mouse model of Alzheimer's diseaseJournal of Neuroinflammation2022
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The Alzheimer’s disease risk factor APOE4 drives pro-inflammation in human astrocytes via HDAC-dependent repression of TAGLN32021
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MT5-MMP is a modulator of inflammation: a study in murine and human neural cells related to Alzheimer’s disease2021
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MT5‐MMP controls APP and β‐CTF/C99 metabolism through proteolytic‐dependent and ‐independent mechanisms relevant for Alzheimer's diseaseFASEB Journal2021
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Human iPSCs-derived astrocytes reveal APOE as a critical modulator of inflammation that could underlie Alzheimer’s disease pathogenesis2021
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MT5-MMP at the crossroads of neuroinflammation and amyloidogenesis: study in murine and human neural cells2021
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MT5-MMP controls APP and β-CTF/C99 metabolism through proteolytic-dependent and -independent mechanisms relevant for Alzheimer’s disease2020
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The influence of vitamin D on neurodegeneration and neurological disorders: a rationale for its physio-pathological actionsCurrent Pharmaceutical Design2020
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High levels of serum soluble TWEAK are associated with neuroinflammation during multiple sclerosisJournal of Translational Medicine2019
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Long-Term Pantethine Treatment Counteracts Pathologic Gene Dysregulation and Decreases Alzheimer’s Disease Pathogenesis in a Transgenic Mouse ModelNeurotherapeutics2019
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Proamyloidogenic effects of membrane type 1 matrix metalloproteinase involve MMP-2 and BACE-1 activities, and the modulation of APP traffickingFASEB Journal2019
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Expression of the Cerebral Olfactory Receptors Olfr110/111 and Olfr544 Is Altered During Aging and in Alzheimer’s Disease-Like MiceMolecular Neurobiology2018
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Differential expression of vitamin D-associated enzymes and receptors in brain cell subtypesJournal of Steroid Biochemistry and Molecular Biology2018
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Chronic treatments with a 5-HT4 receptor agonist decrease amyloid pathology in the entorhinal cortex and learning and memory deficits in the 5xFAD mouse model of Alzheimer's diseaseNeuropharmacology2017
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MT5-MMP Promotes Alzheimer's Pathogenesis in the Frontal Cortex of 5xFAD Mice and APP Trafficking in vitroFrontiers in Molecular Neuroscience2017
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From Blood to Lesioned Brain: An In Vitro Study on Migration Mechanisms of Human Nasal Olfactory Stem CellsStem Cells International2017
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TWEAK-binding autoantibodies are generated during psoriatic arthritis and are not influenced by anti-TNF therapyJournal of Translational Medicine2016
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Is TWEAK a Biomarker for Autoimmune/Chronic Inflammatory Diseases?Frontiers in Immunology2013
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TWEAK/Fn14 pathway modulates properties of a human microvascular endothelial cell model of blood brain barrier.Journal of Neuroinflammation2013
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Cholecalciferol (vitamin D₃) improves myelination and recovery after nerve injuryPLoS ONE2013
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EFFETS DE TWEAK SUR LA BARRIERE HEMATO-ENCEPHALIQUE2012
